Protein kinase C-induced changes in the stoichiometry of ATP binding activate cardiac ATP-sensitive K+ channels. A possible mechanistic link to ischemic preconditioning.
Light, P E
Protein kinase C-induced changes in the stoichiometry of ATP binding activate cardiac ATP-sensitive K+ channels. A possible mechanistic link to ischemic preconditioning. [electronic resource] - Circulation research Sep 1996 - 399-406 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
0009-7330
10.1161/01.res.79.3.399 doi
Adenosine Triphosphate--metabolism
Animals
Chemical Phenomena
Chemistry
Mathematics
Myocardial Ischemia--physiopathology
Myocardial Reperfusion
Myocardium--metabolism
Osmolar Concentration
Patch-Clamp Techniques
Phosphoprotein Phosphatases--pharmacology
Potassium Channels--drug effects
Protein Kinase C--antagonists & inhibitors
Rabbits
Protein kinase C-induced changes in the stoichiometry of ATP binding activate cardiac ATP-sensitive K+ channels. A possible mechanistic link to ischemic preconditioning. [electronic resource] - Circulation research Sep 1996 - 399-406 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
0009-7330
10.1161/01.res.79.3.399 doi
Adenosine Triphosphate--metabolism
Animals
Chemical Phenomena
Chemistry
Mathematics
Myocardial Ischemia--physiopathology
Myocardial Reperfusion
Myocardium--metabolism
Osmolar Concentration
Patch-Clamp Techniques
Phosphoprotein Phosphatases--pharmacology
Potassium Channels--drug effects
Protein Kinase C--antagonists & inhibitors
Rabbits