The potentiation by TNF-alpha and PMA of Fc receptor-mediated phagocytosis in neutrophils is independent of reactive oxygen metabolites produced by NADPH oxidase and of protein kinase C.

Della Bianca, V

The potentiation by TNF-alpha and PMA of Fc receptor-mediated phagocytosis in neutrophils is independent of reactive oxygen metabolites produced by NADPH oxidase and of protein kinase C. [electronic resource] - Biochemical and biophysical research communications Jun 1993 - 919-26 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

0006-291X

10.1006/bbrc.1993.1713 doi


Alkaloids--pharmacology
Dose-Response Relationship, Drug
Humans
Immunoglobulin G--metabolism
In Vitro Techniques
Kinetics
NADH, NADPH Oxidoreductases--antagonists & inhibitors
NADPH Oxidases
Neutrophils--drug effects
Onium Compounds--pharmacology
Phagocytosis--drug effects
Protein Kinase C--antagonists & inhibitors
Receptors, Fc--drug effects
Staurosporine
Superoxide Dismutase--pharmacology
Superoxides--blood
Tetradecanoylphorbol Acetate--pharmacology
Tumor Necrosis Factor-alpha--pharmacology