AML1 fusion transcripts in t(3;21) positive leukemia: evidence of molecular heterogeneity and usage of splicing sites frequently involved in the generation of normal AML1 transcripts.
Sacchi, N
AML1 fusion transcripts in t(3;21) positive leukemia: evidence of molecular heterogeneity and usage of splicing sites frequently involved in the generation of normal AML1 transcripts. [electronic resource] - Genes, chromosomes & cancer Dec 1994 - 226-36 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1045-2257
10.1002/gcc.2870110405 doi
Amino Acid Sequence
Base Sequence
Blast Crisis--genetics
Chromosome Banding
Chromosomes, Human, Pair 21
Chromosomes, Human, Pair 3
Cloning, Molecular
Core Binding Factor Alpha 2 Subunit
DNA Probes
DNA-Binding Proteins
Exons
Humans
In Situ Hybridization, Fluorescence
Karyotyping
Leukemia, Myelogenous, Chronic, BCR-ABL Positive--genetics
Leukemia, Myeloid, Acute--genetics
Molecular Sequence Data
Neoplasm Proteins--chemistry
Proto-Oncogene Proteins
RNA Splicing
RNA, Messenger--genetics
Recombinant Fusion Proteins--chemistry
Transcription Factors
Translocation, Genetic
AML1 fusion transcripts in t(3;21) positive leukemia: evidence of molecular heterogeneity and usage of splicing sites frequently involved in the generation of normal AML1 transcripts. [electronic resource] - Genes, chromosomes & cancer Dec 1994 - 226-36 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1045-2257
10.1002/gcc.2870110405 doi
Amino Acid Sequence
Base Sequence
Blast Crisis--genetics
Chromosome Banding
Chromosomes, Human, Pair 21
Chromosomes, Human, Pair 3
Cloning, Molecular
Core Binding Factor Alpha 2 Subunit
DNA Probes
DNA-Binding Proteins
Exons
Humans
In Situ Hybridization, Fluorescence
Karyotyping
Leukemia, Myelogenous, Chronic, BCR-ABL Positive--genetics
Leukemia, Myeloid, Acute--genetics
Molecular Sequence Data
Neoplasm Proteins--chemistry
Proto-Oncogene Proteins
RNA Splicing
RNA, Messenger--genetics
Recombinant Fusion Proteins--chemistry
Transcription Factors
Translocation, Genetic