The pathogenesis of primary internodal demyelination produced by acetyl ethyl tetramethyl tetralin: evidence for preserved Schwann cell somal function.
Sterman, A B
The pathogenesis of primary internodal demyelination produced by acetyl ethyl tetramethyl tetralin: evidence for preserved Schwann cell somal function. [electronic resource] - Journal of neuropathology and experimental neurology Mar 1981 - 112-21 p. digital
Publication Type: Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
0022-3069
10.1097/00005072-198103000-00004 doi
Animals
Demyelinating Diseases--chemically induced
Myelin Sheath--drug effects
Naphthalenes--poisoning
Nerve Fibers--ultrastructure
Nerve Regeneration
Rats
Schwann Cells--ultrastructure
Tetrahydronaphthalenes--poisoning
Wallerian Degeneration
The pathogenesis of primary internodal demyelination produced by acetyl ethyl tetramethyl tetralin: evidence for preserved Schwann cell somal function. [electronic resource] - Journal of neuropathology and experimental neurology Mar 1981 - 112-21 p. digital
Publication Type: Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
0022-3069
10.1097/00005072-198103000-00004 doi
Animals
Demyelinating Diseases--chemically induced
Myelin Sheath--drug effects
Naphthalenes--poisoning
Nerve Fibers--ultrastructure
Nerve Regeneration
Rats
Schwann Cells--ultrastructure
Tetrahydronaphthalenes--poisoning
Wallerian Degeneration