Curcumin attenuates bevacizumab-induced toxicity via suppressing oxidative stress and preventing mitochondrial dysfunction in heart mitochondria.

Sabet, Nima Shokouhi

Curcumin attenuates bevacizumab-induced toxicity via suppressing oxidative stress and preventing mitochondrial dysfunction in heart mitochondria. [electronic resource] - Naunyn-Schmiedeberg's archives of pharmacology 08 2020 - 1447-1457 p. digital

Publication Type: Journal Article

1432-1912

10.1007/s00210-020-01853-x doi


Angiogenesis Inhibitors--toxicity
Animals
Antioxidants--pharmacology
Apoptosis--drug effects
Bevacizumab--toxicity
Cardiotoxicity
Curcumin--pharmacology
Heart Failure--chemically induced
Lipid Peroxidation--drug effects
Male
Membrane Potential, Mitochondrial--drug effects
Mitochondria, Heart--drug effects
Mitochondrial Swelling--drug effects
Myocytes, Cardiac--drug effects
Oxidative Stress--drug effects
Rats, Wistar
Reactive Oxygen Species--metabolism