GTS-21 ameliorates polymicrobial sepsis-induced hepatic injury by modulating autophagy through α7nAchRs in mice.
Wu, Xiao-Jing
GTS-21 ameliorates polymicrobial sepsis-induced hepatic injury by modulating autophagy through α7nAchRs in mice. [electronic resource] - Cytokine 04 2020 - 155019 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1096-0023
10.1016/j.cyto.2020.155019 doi
Alanine Transaminase--metabolism
Animals
Aspartate Aminotransferases--metabolism
Autophagy--drug effects
Benzylidene Compounds--pharmacology
Cytokines--metabolism
Disease Models, Animal
Inflammation--drug therapy
Interleukin-6--metabolism
Liver--drug effects
Liver Diseases--drug therapy
Male
Mice
Mice, Inbred C57BL
Pyridines--pharmacology
Sepsis--drug therapy
Tumor Necrosis Factor-alpha--metabolism
alpha7 Nicotinic Acetylcholine Receptor--metabolism
GTS-21 ameliorates polymicrobial sepsis-induced hepatic injury by modulating autophagy through α7nAchRs in mice. [electronic resource] - Cytokine 04 2020 - 155019 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1096-0023
10.1016/j.cyto.2020.155019 doi
Alanine Transaminase--metabolism
Animals
Aspartate Aminotransferases--metabolism
Autophagy--drug effects
Benzylidene Compounds--pharmacology
Cytokines--metabolism
Disease Models, Animal
Inflammation--drug therapy
Interleukin-6--metabolism
Liver--drug effects
Liver Diseases--drug therapy
Male
Mice
Mice, Inbred C57BL
Pyridines--pharmacology
Sepsis--drug therapy
Tumor Necrosis Factor-alpha--metabolism
alpha7 Nicotinic Acetylcholine Receptor--metabolism