ACKR2 contributes to pulmonary dysfunction by shaping CCL5:CCR5-dependent recruitment of lymphocytes during influenza A infection in mice.

Tavares, Luciana P

ACKR2 contributes to pulmonary dysfunction by shaping CCL5:CCR5-dependent recruitment of lymphocytes during influenza A infection in mice. [electronic resource] - American journal of physiology. Lung cellular and molecular physiology 04 2020 - L655-L670 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1522-1504

10.1152/ajplung.00134.2019 doi


Animals
B-Lymphocytes--metabolism
Bronchoalveolar Lavage Fluid--virology
Chemokine CCL5--metabolism
Influenza A virus--pathogenicity
Lung--metabolism
Male
Mice
Mice, Inbred C57BL
Mice, Knockout
Orthomyxoviridae Infections--metabolism
Receptors, CCR5--metabolism
Receptors, Chemokine--metabolism
T-Lymphocytes, Regulatory--metabolism