IL-22 and its receptors are increased in human and experimental COPD and contribute to pathogenesis.
Starkey, Malcolm R
IL-22 and its receptors are increased in human and experimental COPD and contribute to pathogenesis. [electronic resource] - The European respiratory journal 07 2019
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1399-3003
10.1183/13993003.00174-2018 doi
Airway Remodeling
Airway Resistance
Animals
Emphysema--etiology
Female
Humans
Immunity, Innate
Interleukins--physiology
Lymphocytes--metabolism
Mice
Mice, Inbred C57BL
Mice, Knockout
Pulmonary Disease, Chronic Obstructive--chemically induced
Receptors, Interleukin--physiology
Smoke--adverse effects
Tobacco Products
Interleukin-22
IL-22 and its receptors are increased in human and experimental COPD and contribute to pathogenesis. [electronic resource] - The European respiratory journal 07 2019
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1399-3003
10.1183/13993003.00174-2018 doi
Airway Remodeling
Airway Resistance
Animals
Emphysema--etiology
Female
Humans
Immunity, Innate
Interleukins--physiology
Lymphocytes--metabolism
Mice
Mice, Inbred C57BL
Mice, Knockout
Pulmonary Disease, Chronic Obstructive--chemically induced
Receptors, Interleukin--physiology
Smoke--adverse effects
Tobacco Products
Interleukin-22