TWEAK/Fn14 Is Overexpressed in Crohn's Disease and Mediates Experimental Ileitis by Regulating Critical Innate and Adaptive Immune Pathways.

Di Martino, Luca

TWEAK/Fn14 Is Overexpressed in Crohn's Disease and Mediates Experimental Ileitis by Regulating Critical Innate and Adaptive Immune Pathways. [electronic resource] - Cellular and molecular gastroenterology and hepatology 2019 - 427-446 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't

2352-345X

10.1016/j.jcmgh.2019.05.009 doi


Adaptive Immunity
Animals
Case-Control Studies
Colitis, Ulcerative--genetics
Crohn Disease--genetics
Cytokine TWEAK--genetics
Disease Models, Animal
Female
Humans
Immunity, Innate
Male
Mice
Middle Aged
TWEAK Receptor--genetics
Up-Regulation