Functional iron deficiency in toxic milk mutant mice (tx-J) despite high hepatic ferroportin: a critical role of decreased GPI-ceruloplasmin expression in liver macrophages.

Jończy, Aneta

Functional iron deficiency in toxic milk mutant mice (tx-J) despite high hepatic ferroportin: a critical role of decreased GPI-ceruloplasmin expression in liver macrophages. [electronic resource] - Metallomics : integrated biometal science 06 2019 - 1079-1092 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1756-591X

10.1039/c9mt00035f doi


Anemia, Iron-Deficiency--etiology
Animals
Apoproteins--metabolism
Cation Transport Proteins--metabolism
Ceruloplasmin--metabolism
Copper--metabolism
Copper-Transporting ATPases--genetics
Disease Models, Animal
Hepatolenticular Degeneration--complications
Iron--metabolism
Liver--metabolism
Mice
Mutation, Missense
Ferroportin