The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded Aβ and prion proteins.
Rossi, Marcello
The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded Aβ and prion proteins. [electronic resource] - Acta neuropathologica communications 04 2019 - 53 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
2051-5960
10.1186/s40478-019-0706-6 doi
Aged
Aged, 80 and over
Alzheimer Disease--genetics
Amyloid beta-Peptides--genetics
Cohort Studies
Creutzfeldt-Jakob Syndrome--genetics
Female
Humans
Male
Middle Aged
Prion Proteins--genetics
Tauopathies--genetics
The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded Aβ and prion proteins. [electronic resource] - Acta neuropathologica communications 04 2019 - 53 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
2051-5960
10.1186/s40478-019-0706-6 doi
Aged
Aged, 80 and over
Alzheimer Disease--genetics
Amyloid beta-Peptides--genetics
Cohort Studies
Creutzfeldt-Jakob Syndrome--genetics
Female
Humans
Male
Middle Aged
Prion Proteins--genetics
Tauopathies--genetics