Erinacine Facilitates the Opening of the Mitochondrial Permeability Transition Pore Through the Inhibition of the PI3K/ Akt/GSK-3β Signaling Pathway in Human Hepatocellular Carcinoma.

Zhou, Li-Jie

Erinacine Facilitates the Opening of the Mitochondrial Permeability Transition Pore Through the Inhibition of the PI3K/ Akt/GSK-3β Signaling Pathway in Human Hepatocellular Carcinoma. [electronic resource] - Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology 2018 - 851-867 p. digital

Publication Type: Journal Article; Retracted Publication

1421-9778

10.1159/000494472 doi


Animals
Apoptosis--drug effects
Carcinoma, Hepatocellular--metabolism
Cell Cycle Checkpoints--drug effects
Chromones--pharmacology
Diterpenes--pharmacology
Female
Glycogen Synthase Kinase 3 beta--genetics
Hep G2 Cells
Humans
Liver Neoplasms--metabolism
Male
Membrane Potential, Mitochondrial--drug effects
Mice, Inbred BALB C
Mice, Nude
Middle Aged
Mitochondrial Membrane Transport Proteins--drug effects
Mitochondrial Permeability Transition Pore
Morpholines--pharmacology
Phosphatidylinositol 3-Kinases--genetics
Proto-Oncogene Proteins c-akt--genetics
Signal Transduction--drug effects