Inhibition of N-glycosylation by tunicamycin attenuates cell-cell adhesion via impaired desmosome formation in normal human epidermal keratinocytes.
Jin, Seon-Pil
Inhibition of N-glycosylation by tunicamycin attenuates cell-cell adhesion via impaired desmosome formation in normal human epidermal keratinocytes. [electronic resource] - Bioscience reports 12 2018
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1573-4935
10.1042/BSR20171641 doi
Cell Adhesion--drug effects
Desmoglein 1--metabolism
Desmoglein 3--metabolism
Desmosomes--drug effects
Epidermis--drug effects
Glycosylation--drug effects
Humans
Keratinocytes--drug effects
Metabolic Networks and Pathways--drug effects
Primary Cell Culture
Tunicamycin--pharmacology
Inhibition of N-glycosylation by tunicamycin attenuates cell-cell adhesion via impaired desmosome formation in normal human epidermal keratinocytes. [electronic resource] - Bioscience reports 12 2018
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1573-4935
10.1042/BSR20171641 doi
Cell Adhesion--drug effects
Desmoglein 1--metabolism
Desmoglein 3--metabolism
Desmosomes--drug effects
Epidermis--drug effects
Glycosylation--drug effects
Humans
Keratinocytes--drug effects
Metabolic Networks and Pathways--drug effects
Primary Cell Culture
Tunicamycin--pharmacology