Increased Asics Expression via the Camkii-CREB Pathway in a Novel Mouse Model of Trigeminal Pain.
Wang, Yan
Increased Asics Expression via the Camkii-CREB Pathway in a Novel Mouse Model of Trigeminal Pain. [electronic resource] - Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology 2018 - 568-578 p. digital
Publication Type: Journal Article
1421-9778
10.1159/000488624 doi
Acetic Acid--toxicity
Acid Sensing Ion Channel Blockers--pharmacology
Acid Sensing Ion Channels--genetics
Acrylamides--pharmacology
Amiloride--pharmacology
Animals
Behavior, Animal--drug effects
Bridged Bicyclo Compounds, Heterocyclic--pharmacology
Calcitonin Gene-Related Peptide--metabolism
Calcium-Calmodulin-Dependent Protein Kinase Type 2--metabolism
Cyclic AMP Response Element-Binding Protein--metabolism
Disease Models, Animal
Male
Mice
Mice, Inbred C57BL
Pain--chemically induced
Phosphorylation--drug effects
Proto-Oncogene Proteins c-fos--genetics
TRPV Cation Channels--antagonists & inhibitors
Trigeminal Nucleus, Spinal--metabolism
Increased Asics Expression via the Camkii-CREB Pathway in a Novel Mouse Model of Trigeminal Pain. [electronic resource] - Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology 2018 - 568-578 p. digital
Publication Type: Journal Article
1421-9778
10.1159/000488624 doi
Acetic Acid--toxicity
Acid Sensing Ion Channel Blockers--pharmacology
Acid Sensing Ion Channels--genetics
Acrylamides--pharmacology
Amiloride--pharmacology
Animals
Behavior, Animal--drug effects
Bridged Bicyclo Compounds, Heterocyclic--pharmacology
Calcitonin Gene-Related Peptide--metabolism
Calcium-Calmodulin-Dependent Protein Kinase Type 2--metabolism
Cyclic AMP Response Element-Binding Protein--metabolism
Disease Models, Animal
Male
Mice
Mice, Inbred C57BL
Pain--chemically induced
Phosphorylation--drug effects
Proto-Oncogene Proteins c-fos--genetics
TRPV Cation Channels--antagonists & inhibitors
Trigeminal Nucleus, Spinal--metabolism