Spinal Protein Kinase Mζ Regulates α-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid Receptor Trafficking and Dendritic Spine Plasticity via Kalirin-7 in the Pathogenesis of Remifentanil-induced Postincisional Hyperalgesia in Rats.

Zhang, Linlin

Spinal Protein Kinase Mζ Regulates α-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid Receptor Trafficking and Dendritic Spine Plasticity via Kalirin-7 in the Pathogenesis of Remifentanil-induced Postincisional Hyperalgesia in Rats. [electronic resource] - Anesthesiology 07 2018 - 173-186 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1528-1175

10.1097/ALN.0000000000002190 doi


Analgesics, Opioid--toxicity
Animals
Dendritic Spines--drug effects
Guanine Nucleotide Exchange Factors--biosynthesis
Hyperalgesia--chemically induced
Male
Neuronal Plasticity--drug effects
Organ Culture Techniques
Pain, Postoperative--chemically induced
Protein Kinase C--physiology
Protein Transport--drug effects
Rats
Rats, Sprague-Dawley
Receptors, AMPA--metabolism
Remifentanil--toxicity
Surgical Wound--complications