Baicalin and its metabolites suppresses gluconeogenesis through activation of AMPK or AKT in insulin resistant HepG-2 cells.

Wang, Tao

Baicalin and its metabolites suppresses gluconeogenesis through activation of AMPK or AKT in insulin resistant HepG-2 cells. [electronic resource] - European journal of medicinal chemistry Dec 2017 - 92-100 p. digital

Publication Type: Journal Article

1768-3254

10.1016/j.ejmech.2017.09.049 doi


AMP-Activated Protein Kinases--metabolism
Cell Survival--drug effects
Dose-Response Relationship, Drug
Flavonoids--chemistry
Gluconeogenesis--drug effects
Hep G2 Cells
Humans
Hypoglycemic Agents--chemistry
Insulin Resistance
Insulin-Secreting Cells--drug effects
Molecular Structure
Proto-Oncogene Proteins c-akt--metabolism
Structure-Activity Relationship
Tumor Cells, Cultured