Interleukin-17 limits hypoxia-inducible factor 1α and development of hypoxic granulomas during tuberculosis.

Domingo-Gonzalez, Racquel

Interleukin-17 limits hypoxia-inducible factor 1α and development of hypoxic granulomas during tuberculosis. [electronic resource] - JCI insight 10 2017

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't

2379-3708

10.1172/jci.insight.92973 doi


Adult
Aged
Animals
Cell Hypoxia--immunology
Female
Gene Expression Regulation--immunology
Granuloma--immunology
Humans
Hypoxia-Inducible Factor 1, alpha Subunit--biosynthesis
Inflammation Mediators--metabolism
Interleukin-17--biosynthesis
Male
Mice, Inbred Strains
Middle Aged
RNA, Messenger--genetics
Tuberculosis, Pulmonary--complications
Young Adult