Targeting fatty acid amide hydrolase and transient receptor potential vanilloid-1 simultaneously to modulate colonic motility and visceral sensation in the mouse: A pharmacological intervention with N-arachidonoyl-serotonin (AA-5-HT).

Bashashati, M

Targeting fatty acid amide hydrolase and transient receptor potential vanilloid-1 simultaneously to modulate colonic motility and visceral sensation in the mouse: A pharmacological intervention with N-arachidonoyl-serotonin (AA-5-HT). [electronic resource] - Neurogastroenterology and motility Dec 2017

Publication Type: Journal Article

1365-2982

10.1111/nmo.13148 doi


Amidohydrolases--metabolism
Animals
Arachidonic Acids--pharmacology
Gastrointestinal Motility--drug effects
Male
Mice
Myenteric Plexus--drug effects
Serotonin--analogs & derivatives
TRPV Cation Channels--metabolism
Visceral Pain--metabolism