Human somatic cells deficient for RAD52 are impaired for viral integration and compromised for most aspects of homology-directed repair.
Kan, Yinan
Human somatic cells deficient for RAD52 are impaired for viral integration and compromised for most aspects of homology-directed repair. [electronic resource] - DNA repair 07 2017 - 64-75 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural
1568-7856
10.1016/j.dnarep.2017.04.006 doi
DNA--metabolism
DNA Breaks, Double-Stranded
DNA Repair
Gene Expression Regulation, Neoplastic
Gene Knockout Techniques
Genomic Instability
Humans
Neoplasms--genetics
Rad52 DNA Repair and Recombination Protein--genetics
Human somatic cells deficient for RAD52 are impaired for viral integration and compromised for most aspects of homology-directed repair. [electronic resource] - DNA repair 07 2017 - 64-75 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural
1568-7856
10.1016/j.dnarep.2017.04.006 doi
DNA--metabolism
DNA Breaks, Double-Stranded
DNA Repair
Gene Expression Regulation, Neoplastic
Gene Knockout Techniques
Genomic Instability
Humans
Neoplasms--genetics
Rad52 DNA Repair and Recombination Protein--genetics