Depletion of pro-oncogenic RUNX2 enhances gemcitabine (GEM) sensitivity of p53-mutated pancreatic cancer Panc-1 cells through the induction of pro-apoptotic TAp63.
Ozaki, Toshinori
Depletion of pro-oncogenic RUNX2 enhances gemcitabine (GEM) sensitivity of p53-mutated pancreatic cancer Panc-1 cells through the induction of pro-apoptotic TAp63. [electronic resource] - Oncotarget Nov 2016 - 71937-71950 p. digital
Publication Type: Journal Article
1949-2553
10.18632/oncotarget.12433 doi
Antimetabolites, Antineoplastic--pharmacology
Apoptosis--drug effects
Cell Cycle Checkpoints
Cell Line, Tumor
Core Binding Factor Alpha 1 Subunit--physiology
Deoxycytidine--analogs & derivatives
Drug Resistance, Neoplasm
Genes, p53
Humans
Membrane Proteins--physiology
Mutation
Pancreatic Neoplasms--drug therapy
Gemcitabine
Depletion of pro-oncogenic RUNX2 enhances gemcitabine (GEM) sensitivity of p53-mutated pancreatic cancer Panc-1 cells through the induction of pro-apoptotic TAp63. [electronic resource] - Oncotarget Nov 2016 - 71937-71950 p. digital
Publication Type: Journal Article
1949-2553
10.18632/oncotarget.12433 doi
Antimetabolites, Antineoplastic--pharmacology
Apoptosis--drug effects
Cell Cycle Checkpoints
Cell Line, Tumor
Core Binding Factor Alpha 1 Subunit--physiology
Deoxycytidine--analogs & derivatives
Drug Resistance, Neoplasm
Genes, p53
Humans
Membrane Proteins--physiology
Mutation
Pancreatic Neoplasms--drug therapy
Gemcitabine