Abandoning M1/M2 for a Network Model of Macrophage Function.

Nahrendorf, Matthias

Abandoning M1/M2 for a Network Model of Macrophage Function. [electronic resource] - Circulation research 07 2016 - 414-7 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Review

1524-4571

10.1161/CIRCRESAHA.116.309194 doi


Animals
Galectin 3--physiology
Humans
Macrophage-1 Antigen--physiology
Macrophages--physiology
Models, Biological
Myocardial Infarction--blood