Cu(II)-coumestrol interaction leads to ROS-mediated DNA damage and cell death: a putative mechanism for anticancer activity.
Zafar, Atif
Cu(II)-coumestrol interaction leads to ROS-mediated DNA damage and cell death: a putative mechanism for anticancer activity. [electronic resource] - The Journal of nutritional biochemistry 07 2016 - 15-27 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1873-4847
10.1016/j.jnutbio.2016.03.003 doi
Anticarcinogenic Agents--chemistry
Apoptosis--drug effects
Biomarkers--metabolism
Cell Survival
Cells, Cultured
Chelating Agents--pharmacology
Comet Assay
Copper--chemistry
Coumestrol--antagonists & inhibitors
DNA Fragmentation--drug effects
Dietary Supplements
Free Radical Scavengers--pharmacology
Humans
Lipid Peroxidation--drug effects
Lymphocytes--cytology
Microscopy, Electron, Scanning
Oxidative Stress--drug effects
Phytoestrogens--antagonists & inhibitors
Protein Carbonylation--drug effects
Reactive Oxygen Species--agonists
Cu(II)-coumestrol interaction leads to ROS-mediated DNA damage and cell death: a putative mechanism for anticancer activity. [electronic resource] - The Journal of nutritional biochemistry 07 2016 - 15-27 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1873-4847
10.1016/j.jnutbio.2016.03.003 doi
Anticarcinogenic Agents--chemistry
Apoptosis--drug effects
Biomarkers--metabolism
Cell Survival
Cells, Cultured
Chelating Agents--pharmacology
Comet Assay
Copper--chemistry
Coumestrol--antagonists & inhibitors
DNA Fragmentation--drug effects
Dietary Supplements
Free Radical Scavengers--pharmacology
Humans
Lipid Peroxidation--drug effects
Lymphocytes--cytology
Microscopy, Electron, Scanning
Oxidative Stress--drug effects
Phytoestrogens--antagonists & inhibitors
Protein Carbonylation--drug effects
Reactive Oxygen Species--agonists