Environmentally Persistent Free Radicals Cause Apoptosis in HL-1 Cardiomyocytes.
Chuang, Gin C
Environmentally Persistent Free Radicals Cause Apoptosis in HL-1 Cardiomyocytes. [electronic resource] - Cardiovascular toxicology 04 2017 - 140-149 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural
1559-0259
10.1007/s12012-016-9367-x doi
Animals
Antioxidants--pharmacology
Apoptosis--drug effects
Caspases--metabolism
Cell Line
Dose-Response Relationship, Drug
Environmental Pollutants--toxicity
Free Radicals--toxicity
Male
Membrane Potential, Mitochondrial--drug effects
Mice
Mitochondria--drug effects
Myocytes, Cardiac--drug effects
Oxidative Stress--drug effects
Particulate Matter--toxicity
Poly(ADP-ribose) Polymerases--metabolism
Rats
Rats, Sprague-Dawley
Signal Transduction--drug effects
Time Factors
Environmentally Persistent Free Radicals Cause Apoptosis in HL-1 Cardiomyocytes. [electronic resource] - Cardiovascular toxicology 04 2017 - 140-149 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural
1559-0259
10.1007/s12012-016-9367-x doi
Animals
Antioxidants--pharmacology
Apoptosis--drug effects
Caspases--metabolism
Cell Line
Dose-Response Relationship, Drug
Environmental Pollutants--toxicity
Free Radicals--toxicity
Male
Membrane Potential, Mitochondrial--drug effects
Mice
Mitochondria--drug effects
Myocytes, Cardiac--drug effects
Oxidative Stress--drug effects
Particulate Matter--toxicity
Poly(ADP-ribose) Polymerases--metabolism
Rats
Rats, Sprague-Dawley
Signal Transduction--drug effects
Time Factors