Transmembrane TNF-α is sufficient for articular inflammation and hypernociception in a mouse model of gout.
Amaral, Flávio A
Transmembrane TNF-α is sufficient for articular inflammation and hypernociception in a mouse model of gout. [electronic resource] - European journal of immunology Jan 2016 - 204-11 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1521-4141
10.1002/eji.201545798 doi
Animals
Disease Models, Animal
Gout--complications
Hyperalgesia--etiology
Inflammation--immunology
Interleukin-1beta--immunology
Knee Joint
Male
Membrane Proteins--metabolism
Mice
Mice, Inbred C57BL
Mice, Transgenic
Physical Stimulation
Real-Time Polymerase Chain Reaction
Tumor Necrosis Factor-alpha--metabolism
Uric Acid--adverse effects
Transmembrane TNF-α is sufficient for articular inflammation and hypernociception in a mouse model of gout. [electronic resource] - European journal of immunology Jan 2016 - 204-11 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1521-4141
10.1002/eji.201545798 doi
Animals
Disease Models, Animal
Gout--complications
Hyperalgesia--etiology
Inflammation--immunology
Interleukin-1beta--immunology
Knee Joint
Male
Membrane Proteins--metabolism
Mice
Mice, Inbred C57BL
Mice, Transgenic
Physical Stimulation
Real-Time Polymerase Chain Reaction
Tumor Necrosis Factor-alpha--metabolism
Uric Acid--adverse effects