Hypoxia defined as a common culprit/initiation factor in mitochondrial-mediated proinflammatory processes.
Stefano, George B
Hypoxia defined as a common culprit/initiation factor in mitochondrial-mediated proinflammatory processes. [electronic resource] - Medical science monitor : international medical journal of experimental and clinical research May 2015 - 1478-84 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Review
1643-3750
10.12659/MSM.894437 doi
Adenosine Triphosphate--biosynthesis
Animals
Biological Evolution
Cell Hypoxia--physiology
Cellular Microenvironment
Cellular Senescence
Cytokines--metabolism
Energy Metabolism
Gene Expression Regulation
Humans
Immunity, Innate
Immunocompetence
Inflammation--etiology
Invertebrates--physiology
Microglia--physiology
Mitochondria--physiology
Models, Biological
Neuroimmunomodulation--physiology
Nitric Oxide--physiology
Nitrites--metabolism
Opioid Peptides--metabolism
Signal Transduction
Vertebrates--physiology
Hypoxia defined as a common culprit/initiation factor in mitochondrial-mediated proinflammatory processes. [electronic resource] - Medical science monitor : international medical journal of experimental and clinical research May 2015 - 1478-84 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Review
1643-3750
10.12659/MSM.894437 doi
Adenosine Triphosphate--biosynthesis
Animals
Biological Evolution
Cell Hypoxia--physiology
Cellular Microenvironment
Cellular Senescence
Cytokines--metabolism
Energy Metabolism
Gene Expression Regulation
Humans
Immunity, Innate
Immunocompetence
Inflammation--etiology
Invertebrates--physiology
Microglia--physiology
Mitochondria--physiology
Models, Biological
Neuroimmunomodulation--physiology
Nitric Oxide--physiology
Nitrites--metabolism
Opioid Peptides--metabolism
Signal Transduction
Vertebrates--physiology