TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain.
Wang, Lan-Wan
TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain. [electronic resource] - Journal of neuroinflammation Dec 2014 - 215 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1742-2094
10.1186/s12974-014-0215-2 doi
Animals
Animals, Newborn
Blood-Brain Barrier--drug effects
Brain--drug effects
Female
Hypoxia-Ischemia, Brain--chemically induced
Inflammation--chemically induced
Lipopolysaccharides--toxicity
MAP Kinase Signaling System--drug effects
Male
Mice
Mice, 129 Strain
Mice, Inbred C57BL
Mice, Knockout
Receptors, Tumor Necrosis Factor, Type I--biosynthesis
TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain. [electronic resource] - Journal of neuroinflammation Dec 2014 - 215 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1742-2094
10.1186/s12974-014-0215-2 doi
Animals
Animals, Newborn
Blood-Brain Barrier--drug effects
Brain--drug effects
Female
Hypoxia-Ischemia, Brain--chemically induced
Inflammation--chemically induced
Lipopolysaccharides--toxicity
MAP Kinase Signaling System--drug effects
Male
Mice
Mice, 129 Strain
Mice, Inbred C57BL
Mice, Knockout
Receptors, Tumor Necrosis Factor, Type I--biosynthesis