GSK-3β dysregulation contributes to parkinson's-like pathophysiology with associated region-specific phosphorylation and accumulation of tau and α-synuclein.
Credle, J J
GSK-3β dysregulation contributes to parkinson's-like pathophysiology with associated region-specific phosphorylation and accumulation of tau and α-synuclein. [electronic resource] - Cell death and differentiation May 2015 - 838-51 p. digital
Publication Type: Journal Article
1476-5403
10.1038/cdd.2014.179 doi
Animals
Glycogen Synthase Kinase 3--genetics
Glycogen Synthase Kinase 3 beta
Humans
Mice
Mice, Transgenic
Parkinsonian Disorders--genetics
alpha-Synuclein--genetics
tau Proteins--genetics
GSK-3β dysregulation contributes to parkinson's-like pathophysiology with associated region-specific phosphorylation and accumulation of tau and α-synuclein. [electronic resource] - Cell death and differentiation May 2015 - 838-51 p. digital
Publication Type: Journal Article
1476-5403
10.1038/cdd.2014.179 doi
Animals
Glycogen Synthase Kinase 3--genetics
Glycogen Synthase Kinase 3 beta
Humans
Mice
Mice, Transgenic
Parkinsonian Disorders--genetics
alpha-Synuclein--genetics
tau Proteins--genetics