Inactivation of Uaf1 causes defective homologous recombination and early embryonic lethality in mice.

Park, Eunmi

Inactivation of Uaf1 causes defective homologous recombination and early embryonic lethality in mice. [electronic resource] - Molecular and cellular biology Nov 2013 - 4360-70 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't

1098-5549

10.1128/MCB.00870-13 doi


Animals
Carcinogenesis--genetics
Cells, Cultured
Chromosomal Instability
DNA Repair
Embryo Loss--genetics
Embryonic Stem Cells--drug effects
Fanconi Anemia Complementation Group D2 Protein--metabolism
Female
Fibroblasts--drug effects
Gene Deletion
Homologous Recombination
Homozygote
Humans
Infertility--genetics
Male
Mice--embryology
Mice, Inbred C57BL
Mutagens--pharmacology
Nuclear Proteins--genetics
Proliferating Cell Nuclear Antigen--metabolism
Ubiquitination