Increased radial glia quiescence, decreased reactivation upon injury and unaltered neuroblast behavior underlie decreased neurogenesis in the aging zebrafish telencephalon.

Edelmann, Kathrin

Increased radial glia quiescence, decreased reactivation upon injury and unaltered neuroblast behavior underlie decreased neurogenesis in the aging zebrafish telencephalon. [electronic resource] - The Journal of comparative neurology Sep 2013 - 3099-115 p. digital

Publication Type: Journal Article

1096-9861

10.1002/cne.23347 doi


Age Factors
Aging
Animals
Animals, Genetically Modified
Brain Injuries--pathology
Bromodeoxyuridine--metabolism
Cell Count
Disease Models, Animal
ELAV Proteins--genetics
ELAV-Like Protein 3
Green Fluorescent Proteins--genetics
Histones--metabolism
Nerve Regeneration--physiology
Nerve Tissue Proteins--metabolism
Neural Stem Cells--physiology
Neuroglia--physiology
Proliferating Cell Nuclear Antigen--metabolism
S100 Calcium Binding Protein beta Subunit--metabolism
Telencephalon--pathology
Zebrafish
Zebrafish Proteins--genetics