Class IA PI3K p110β subunit promotes autophagy through Rab5 small GTPase in response to growth factor limitation.
Dou, Zhixun
Class IA PI3K p110β subunit promotes autophagy through Rab5 small GTPase in response to growth factor limitation. [electronic resource] - Molecular cell Apr 2013 - 29-42 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
1097-4164
10.1016/j.molcel.2013.01.022 doi
Animals
Autophagy
Class I Phosphatidylinositol 3-Kinases--deficiency
Class III Phosphatidylinositol 3-Kinases--metabolism
Guanosine Triphosphate--metabolism
HEK293 Cells
Humans
Intercellular Signaling Peptides and Proteins--deficiency
Mice
Mice, Knockout
Mutation
Phosphatidylinositol 3-Kinases--deficiency
Signal Transduction
Transfection
rab5 GTP-Binding Proteins--metabolism
Class IA PI3K p110β subunit promotes autophagy through Rab5 small GTPase in response to growth factor limitation. [electronic resource] - Molecular cell Apr 2013 - 29-42 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
1097-4164
10.1016/j.molcel.2013.01.022 doi
Animals
Autophagy
Class I Phosphatidylinositol 3-Kinases--deficiency
Class III Phosphatidylinositol 3-Kinases--metabolism
Guanosine Triphosphate--metabolism
HEK293 Cells
Humans
Intercellular Signaling Peptides and Proteins--deficiency
Mice
Mice, Knockout
Mutation
Phosphatidylinositol 3-Kinases--deficiency
Signal Transduction
Transfection
rab5 GTP-Binding Proteins--metabolism