Deletion of Fn14 receptor protects from right heart fibrosis and dysfunction.
Novoyatleva, Tatyana
Deletion of Fn14 receptor protects from right heart fibrosis and dysfunction. [electronic resource] - Basic research in cardiology Mar 2013 - 325 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1435-1803
10.1007/s00395-012-0325-x doi
Animals
Apoptosis Regulatory Proteins--genetics
Blotting, Western
Cell Differentiation
Cell Proliferation
Collagen--metabolism
Cytokine TWEAK
Endothelin-1--physiology
Familial Primary Pulmonary Hypertension
Fibrosis--prevention & control
Fluorescent Antibody Technique
Hypertension, Pulmonary--complications
Hypertrophy, Right Ventricular--metabolism
Immunohistochemistry
Membrane Proteins--genetics
Mice
Mice, Knockout
Myocardium--pathology
Myofibroblasts
Rats
Rats, Sprague-Dawley
Real-Time Polymerase Chain Reaction
Receptors, Tumor Necrosis Factor--physiology
Signal Transduction
TWEAK Receptor
Trans-Activators--genetics
Transcription Factors--genetics
Tumor Necrosis Factors--genetics
Up-Regulation
Ventricular Dysfunction, Right--metabolism
Deletion of Fn14 receptor protects from right heart fibrosis and dysfunction. [electronic resource] - Basic research in cardiology Mar 2013 - 325 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1435-1803
10.1007/s00395-012-0325-x doi
Animals
Apoptosis Regulatory Proteins--genetics
Blotting, Western
Cell Differentiation
Cell Proliferation
Collagen--metabolism
Cytokine TWEAK
Endothelin-1--physiology
Familial Primary Pulmonary Hypertension
Fibrosis--prevention & control
Fluorescent Antibody Technique
Hypertension, Pulmonary--complications
Hypertrophy, Right Ventricular--metabolism
Immunohistochemistry
Membrane Proteins--genetics
Mice
Mice, Knockout
Myocardium--pathology
Myofibroblasts
Rats
Rats, Sprague-Dawley
Real-Time Polymerase Chain Reaction
Receptors, Tumor Necrosis Factor--physiology
Signal Transduction
TWEAK Receptor
Trans-Activators--genetics
Transcription Factors--genetics
Tumor Necrosis Factors--genetics
Up-Regulation
Ventricular Dysfunction, Right--metabolism