Anti-chlamydial Th17 responses are controlled by the inducible costimulator partially through phosphoinositide 3-kinase signaling.
Gao, Xiaoling
Anti-chlamydial Th17 responses are controlled by the inducible costimulator partially through phosphoinositide 3-kinase signaling. [electronic resource] - PloS one 2012 - e52657 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1932-6203
10.1371/journal.pone.0052657 doi
Animals
Antibodies, Bacterial--immunology
Antibody Formation--genetics
Bacterial Load
Chlamydia Infections--immunology
Chlamydia muridarum--immunology
Disease Models, Animal
Female
Inducible T-Cell Co-Stimulator Protein--genetics
Interferon-gamma--biosynthesis
Lung--immunology
Mice
Phosphatidylinositol 3-Kinases--metabolism
Signal Transduction
Spleen--immunology
Th17 Cells--immunology
Anti-chlamydial Th17 responses are controlled by the inducible costimulator partially through phosphoinositide 3-kinase signaling. [electronic resource] - PloS one 2012 - e52657 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1932-6203
10.1371/journal.pone.0052657 doi
Animals
Antibodies, Bacterial--immunology
Antibody Formation--genetics
Bacterial Load
Chlamydia Infections--immunology
Chlamydia muridarum--immunology
Disease Models, Animal
Female
Inducible T-Cell Co-Stimulator Protein--genetics
Interferon-gamma--biosynthesis
Lung--immunology
Mice
Phosphatidylinositol 3-Kinases--metabolism
Signal Transduction
Spleen--immunology
Th17 Cells--immunology