Mitochondria malfunctions as mediators of stem-cells' related carcinogenesis: a hypothesis that supports the highly conserved profile of carcinogenesis.

Hatzi, Vasiliki I

Mitochondria malfunctions as mediators of stem-cells' related carcinogenesis: a hypothesis that supports the highly conserved profile of carcinogenesis. [electronic resource] - Medical hypotheses Jan 2013 - 70-4 p. digital

Publication Type: Journal Article

1532-2777

10.1016/j.mehy.2012.10.002 doi


Adenosine Triphosphate--metabolism
Apoptosis--physiology
Autophagy--physiology
Cell Transformation, Neoplastic--metabolism
Energy Metabolism--physiology
Humans
Mitochondria--physiology
Models, Biological
Neoplasms--physiopathology
Neoplastic Stem Cells--physiology