[The changes of metabolism in myocardium at ischemia-reperfusion and activating of the ATP-sensitive potassium channels].
Strutyns'kyĭ, R B
[The changes of metabolism in myocardium at ischemia-reperfusion and activating of the ATP-sensitive potassium channels]. [electronic resource] - Fiziolohichnyi zhurnal (Kiev, Ukraine : 1994) 2012 - 13-26 p. digital
Publication Type: Journal Article
2522-9028
Adenosine Triphosphate--metabolism
Animals
Apoptosis--drug effects
Arachidonic Acid--antagonists & inhibitors
Arginase--antagonists & inhibitors
Arginine--metabolism
Dogs
Eicosanoids--antagonists & inhibitors
Guanosine Triphosphate--metabolism
Heme
KATP Channels--agonists
Mitochondria, Heart--drug effects
Mitochondrial Membrane Transport Proteins--antagonists & inhibitors
Mitochondrial Permeability Transition Pore
Myocardium--metabolism
Nitrate Reductase (NADH)--metabolism
Nitric Oxide--agonists
Nitric Oxide Synthase Type I--metabolism
Nitric Oxide Synthase Type II--metabolism
Pinacidil--administration & dosage
Reactive Nitrogen Species--metabolism
Reactive Oxygen Species--metabolism
Reperfusion Injury--drug therapy
[The changes of metabolism in myocardium at ischemia-reperfusion and activating of the ATP-sensitive potassium channels]. [electronic resource] - Fiziolohichnyi zhurnal (Kiev, Ukraine : 1994) 2012 - 13-26 p. digital
Publication Type: Journal Article
2522-9028
Adenosine Triphosphate--metabolism
Animals
Apoptosis--drug effects
Arachidonic Acid--antagonists & inhibitors
Arginase--antagonists & inhibitors
Arginine--metabolism
Dogs
Eicosanoids--antagonists & inhibitors
Guanosine Triphosphate--metabolism
Heme
KATP Channels--agonists
Mitochondria, Heart--drug effects
Mitochondrial Membrane Transport Proteins--antagonists & inhibitors
Mitochondrial Permeability Transition Pore
Myocardium--metabolism
Nitrate Reductase (NADH)--metabolism
Nitric Oxide--agonists
Nitric Oxide Synthase Type I--metabolism
Nitric Oxide Synthase Type II--metabolism
Pinacidil--administration & dosage
Reactive Nitrogen Species--metabolism
Reactive Oxygen Species--metabolism
Reperfusion Injury--drug therapy