A common cellular pathway for v-mos and v-Ki-ras is not required for v-Ki-ras-induced tumorigenicity in a nonmalignant, v-mos-expressing revertant cell.

Gerfaux, J

A common cellular pathway for v-mos and v-Ki-ras is not required for v-Ki-ras-induced tumorigenicity in a nonmalignant, v-mos-expressing revertant cell. [electronic resource] - Molecular carcinogenesis 1990 - 103-13 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

0899-1987

10.1002/mc.2940030208 doi


Animals
Cell Transformation, Viral
Chimera
Collagen--biosynthesis
Down-Regulation
Mice
Mice, Inbred BALB C
Mice, Nude
Moloney murine sarcoma virus--genetics
Oncogene Proteins v-mos
Oncogenes
Promoter Regions, Genetic
Protein-Tyrosine Kinases--genetics
Proto-Oncogene Proteins--genetics
Proto-Oncogene Proteins p21(ras)
Retroviridae Proteins, Oncogenic--genetics
Sarcoma Viruses, Murine--genetics
Tumor Cells, Cultured