Traumatic brain injury and trichloroethylene exposure interact and produce functional, histological, and mitochondrial deficits.

Sauerbeck, Andrew

Traumatic brain injury and trichloroethylene exposure interact and produce functional, histological, and mitochondrial deficits. [electronic resource] - Experimental neurology Mar 2012 - 85-94 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural

1090-2430

10.1016/j.expneurol.2011.12.012 doi


Animals
Brain Injuries--complications
Corpus Striatum--pathology
Disease Models, Animal
Dopamine Plasma Membrane Transport Proteins--metabolism
Gene Expression Regulation--drug effects
Male
Mitochondrial Diseases--etiology
Motor Activity--drug effects
Movement Disorders--etiology
Oxygen Consumption--drug effects
Rats
Rats, Inbred F344
Receptors, Dopamine D2--metabolism
Rotarod Performance Test
Solvents--toxicity
Trichloroethylene--toxicity
Tyrosine 3-Monooxygenase--metabolism