Impaired TGF-β signaling and a defect in resolution of inflammation contribute to delayed wound healing in a female rat model of type 2 diabetes.
Al-Mulla, Fahd
Impaired TGF-β signaling and a defect in resolution of inflammation contribute to delayed wound healing in a female rat model of type 2 diabetes. [electronic resource] - Molecular bioSystems Nov 2011 - 3006-20 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1742-2051
10.1039/c0mb00317d doi
Animals
Cell Line
Cell Movement
Diabetes Mellitus, Type 2--immunology
Estrogens--metabolism
Female
Fibroblasts--pathology
Humans
Inflammation--metabolism
Macrophages--pathology
NF-kappa B--physiology
Oxidative Stress
Rats
Rats, Wistar
Signal Transduction
Skin--cytology
Smad2 Protein--metabolism
Smad7 Protein--metabolism
Transforming Growth Factor beta--metabolism
Tumor Necrosis Factor-alpha--physiology
Wound Healing
Impaired TGF-β signaling and a defect in resolution of inflammation contribute to delayed wound healing in a female rat model of type 2 diabetes. [electronic resource] - Molecular bioSystems Nov 2011 - 3006-20 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1742-2051
10.1039/c0mb00317d doi
Animals
Cell Line
Cell Movement
Diabetes Mellitus, Type 2--immunology
Estrogens--metabolism
Female
Fibroblasts--pathology
Humans
Inflammation--metabolism
Macrophages--pathology
NF-kappa B--physiology
Oxidative Stress
Rats
Rats, Wistar
Signal Transduction
Skin--cytology
Smad2 Protein--metabolism
Smad7 Protein--metabolism
Transforming Growth Factor beta--metabolism
Tumor Necrosis Factor-alpha--physiology
Wound Healing