FLT3-mediated p38-MAPK activation participates in the control of megakaryopoiesis in primary myelofibrosis.
Desterke, Christophe
FLT3-mediated p38-MAPK activation participates in the control of megakaryopoiesis in primary myelofibrosis. [electronic resource] - Cancer research Apr 2011 - 2901-15 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1538-7445
10.1158/0008-5472.CAN-10-1731 doi
Antigens, CD34--biosynthesis
Enzyme Activation
Hematopoietic Stem Cells--enzymology
Humans
Leukocytes, Mononuclear--enzymology
MAP Kinase Signaling System
Megakaryocytes--enzymology
Membrane Proteins--blood
Mitogen-Activated Protein Kinases--metabolism
Phosphorylation
Primary Myelofibrosis--blood
Stromal Cells--enzymology
fms-Like Tyrosine Kinase 3--biosynthesis
p38 Mitogen-Activated Protein Kinases--metabolism
FLT3-mediated p38-MAPK activation participates in the control of megakaryopoiesis in primary myelofibrosis. [electronic resource] - Cancer research Apr 2011 - 2901-15 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1538-7445
10.1158/0008-5472.CAN-10-1731 doi
Antigens, CD34--biosynthesis
Enzyme Activation
Hematopoietic Stem Cells--enzymology
Humans
Leukocytes, Mononuclear--enzymology
MAP Kinase Signaling System
Megakaryocytes--enzymology
Membrane Proteins--blood
Mitogen-Activated Protein Kinases--metabolism
Phosphorylation
Primary Myelofibrosis--blood
Stromal Cells--enzymology
fms-Like Tyrosine Kinase 3--biosynthesis
p38 Mitogen-Activated Protein Kinases--metabolism