AMP-activated protein kinase inhibits homocysteine-induced dysfunction and apoptosis in endothelial progenitor cells.

Jia, Fang

AMP-activated protein kinase inhibits homocysteine-induced dysfunction and apoptosis in endothelial progenitor cells. [electronic resource] - Cardiovascular drugs and therapy Feb 2011 - 21-9 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1573-7241

10.1007/s10557-010-6277-1 doi


AMP-Activated Protein Kinases--metabolism
Aminoimidazole Carboxamide--analogs & derivatives
Apoptosis--drug effects
Atorvastatin
Cell Movement--drug effects
Cell Proliferation--drug effects
Cells, Cultured
Down-Regulation--drug effects
Endothelial Cells--cytology
Heptanoic Acids--pharmacology
Homocysteine--antagonists & inhibitors
Humans
NADPH Oxidase 4
NADPH Oxidases--metabolism
Nitric Oxide--metabolism
Nitric Oxide Synthase Type III--genetics
Phosphorylation--drug effects
Pyrroles--pharmacology
Reactive Oxygen Species--metabolism
Ribonucleotides--pharmacology
Stem Cells--cytology
Up-Regulation--drug effects