Sustained hemodynamic stress disrupts normal circadian rhythms in calcineurin-dependent signaling and protein phosphorylation in the heart.

Sachan, Nita

Sustained hemodynamic stress disrupts normal circadian rhythms in calcineurin-dependent signaling and protein phosphorylation in the heart. [electronic resource] - Circulation research Feb 2011 - 437-45 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't

1524-4571

10.1161/CIRCRESAHA.110.235309 doi


Animals
Calcineurin--physiology
Calcium-Binding Proteins--metabolism
Circadian Rhythm--physiology
Disease Models, Animal
Heart Failure--metabolism
Heart Ventricles--metabolism
Hemodynamics--physiology
Intracellular Signaling Peptides and Proteins--metabolism
Male
Mice
Mice, Inbred C57BL
Muscle Proteins--metabolism
NFATC Transcription Factors--metabolism
Phosphorylation--physiology
Protein Phosphatase 1--metabolism
Proteins--metabolism
Signal Transduction--physiology
Stress, Physiological--physiology