JS-K, a nitric oxide-releasing prodrug, modulates ß-catenin/TCF signaling in leukemic Jurkat cells: evidence of an S-nitrosylated mechanism.

Nath, Niharika

JS-K, a nitric oxide-releasing prodrug, modulates ß-catenin/TCF signaling in leukemic Jurkat cells: evidence of an S-nitrosylated mechanism. [electronic resource] - Biochemical pharmacology Dec 2010 - 1641-9 p. digital

Publication Type: Comparative Study; Journal Article; Research Support, N.I.H., Extramural

1873-2968

10.1016/j.bcp.2010.08.011 doi


Apoptosis--drug effects
Azo Compounds--pharmacology
Basic Helix-Loop-Helix Leucine Zipper Transcription Factors--physiology
Growth Inhibitors--pharmacology
Humans
Jurkat Cells
Nitric Oxide--metabolism
Nitroso Compounds--pharmacology
Piperazines--pharmacology
Precursor T-Cell Lymphoblastic Leukemia-Lymphoma--drug therapy
Prodrugs--pharmacology
Signal Transduction--drug effects
Transcription Factor 4
Transcription Factors--physiology
Wnt Proteins--physiology
beta Catenin--physiology