A study of long-term potentiation in transgenic mice over-expressing mutant forms of both amyloid precursor protein and presenilin-1.

Fitzjohn, Stephen M

A study of long-term potentiation in transgenic mice over-expressing mutant forms of both amyloid precursor protein and presenilin-1. [electronic resource] - Molecular brain Jul 2010 - 21 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1756-6606

10.1186/1756-6606-3-21 doi


Aging--pathology
Amyloid beta-Protein Precursor--genetics
Animals
CA1 Region, Hippocampal--physiopathology
Electric Stimulation
Excitatory Postsynaptic Potentials--physiology
Humans
Long-Term Potentiation--physiology
Mice
Mice, Transgenic
Mutant Proteins--genetics
Plaque, Amyloid--metabolism
Presenilin-1--genetics
Synaptic Transmission--physiology
Tetany--physiopathology