A single mutation promotes amyloidogenicity through a highly promiscuous dimer interface.

Peterson, Francis C

A single mutation promotes amyloidogenicity through a highly promiscuous dimer interface. [electronic resource] - Structure (London, England : 1993) May 2010 - 563-70 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural

1878-4186

10.1016/j.str.2010.02.012 doi


Amyloid--chemistry
Amyloidosis--genetics
Humans
Mutation
Proteostasis Deficiencies