APA

Zhao J. T., Hill A. P., Varghese A., Cooper A. A., Swan H., Laitinen-Forsblom P. J., Rees M. I., Skinner J. R., Campbell T. J. & Vandenberg J. I. (20100426). Not all hERG pore domain mutations have a severe phenotype: G584S has an inactivation gating defect with mild phenotype compared to G572S, which has a dominant negative trafficking defect and a severe phenotype. : Journal of cardiovascular electrophysiology.

Chicago

Zhao Jing Ting, Hill Adam P, Varghese Anthony, Cooper Antony A, Swan Heikki, Laitinen-Forsblom Päivi J, Rees Mark I, Skinner Jonathan R, Campbell Terence J and Vandenberg Jamie I. 20100426. Not all hERG pore domain mutations have a severe phenotype: G584S has an inactivation gating defect with mild phenotype compared to G572S, which has a dominant negative trafficking defect and a severe phenotype. : Journal of cardiovascular electrophysiology.

Harvard

Zhao J. T., Hill A. P., Varghese A., Cooper A. A., Swan H., Laitinen-Forsblom P. J., Rees M. I., Skinner J. R., Campbell T. J. and Vandenberg J. I. (20100426). Not all hERG pore domain mutations have a severe phenotype: G584S has an inactivation gating defect with mild phenotype compared to G572S, which has a dominant negative trafficking defect and a severe phenotype. : Journal of cardiovascular electrophysiology.

MLA

Zhao Jing Ting, Hill Adam P, Varghese Anthony, Cooper Antony A, Swan Heikki, Laitinen-Forsblom Päivi J, Rees Mark I, Skinner Jonathan R, Campbell Terence J and Vandenberg Jamie I. Not all hERG pore domain mutations have a severe phenotype: G584S has an inactivation gating defect with mild phenotype compared to G572S, which has a dominant negative trafficking defect and a severe phenotype. : Journal of cardiovascular electrophysiology. 20100426.