Loss of biphasic effect on Na/K-ATPase activity by angiotensin II involves defective angiotensin type 1 receptor-nitric oxide signaling.

Banday, Anees Ahmad

Loss of biphasic effect on Na/K-ATPase activity by angiotensin II involves defective angiotensin type 1 receptor-nitric oxide signaling. [electronic resource] - Hypertension (Dallas, Tex. : 1979) Dec 2008 - 1099-105 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't

1524-4563

10.1161/HYPERTENSIONAHA.108.117911 doi


Angiotensin II--pharmacology
Animals
Antihypertensive Agents--pharmacology
Antioxidants--pharmacology
Benzimidazoles--pharmacology
Biphenyl Compounds
Blood Pressure--drug effects
Buthionine Sulfoximine--pharmacology
Cyclic GMP--metabolism
Cyclic N-Oxides--pharmacology
Enzyme Inhibitors--pharmacology
Focal Adhesion Kinase 2--metabolism
Hypertension, Renal--chemically induced
Kidney Tubules, Proximal--drug effects
Male
Mitogen-Activated Protein Kinases--metabolism
Nitric Oxide--metabolism
Oxidative Stress--drug effects
Rats
Rats, Sprague-Dawley
Receptor, Angiotensin, Type 1--metabolism
Signal Transduction--drug effects
Sodium-Hydrogen Exchanger 3
Sodium-Hydrogen Exchangers--metabolism
Sodium-Potassium-Exchanging ATPase--metabolism
Spin Labels
Tetrazoles--pharmacology
Vasoconstrictor Agents--pharmacology