Selective cyclooxygenase-2 inhibition directly increases human vascular reactivity to norepinephrine during acute inflammation.
Foudi, Nabil
Selective cyclooxygenase-2 inhibition directly increases human vascular reactivity to norepinephrine during acute inflammation. [electronic resource] - Cardiovascular research Feb 2009 - 269-77 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1755-3245
10.1093/cvr/cvn287 doi
6-Ketoprostaglandin F1 alpha--analogs & derivatives
Acute Disease
Aged
Cyclooxygenase 1--analysis
Cyclooxygenase 2--analysis
Cyclooxygenase 2 Inhibitors--pharmacology
Dinoprostone--metabolism
Endothelium, Vascular--physiology
Female
Humans
Inflammation--physiopathology
Interleukin-1beta--pharmacology
Lipopolysaccharides--pharmacology
Male
Middle Aged
Norepinephrine--pharmacology
Organ Culture Techniques
Thromboxane B2--metabolism
Vasoconstriction--drug effects
Selective cyclooxygenase-2 inhibition directly increases human vascular reactivity to norepinephrine during acute inflammation. [electronic resource] - Cardiovascular research Feb 2009 - 269-77 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1755-3245
10.1093/cvr/cvn287 doi
6-Ketoprostaglandin F1 alpha--analogs & derivatives
Acute Disease
Aged
Cyclooxygenase 1--analysis
Cyclooxygenase 2--analysis
Cyclooxygenase 2 Inhibitors--pharmacology
Dinoprostone--metabolism
Endothelium, Vascular--physiology
Female
Humans
Inflammation--physiopathology
Interleukin-1beta--pharmacology
Lipopolysaccharides--pharmacology
Male
Middle Aged
Norepinephrine--pharmacology
Organ Culture Techniques
Thromboxane B2--metabolism
Vasoconstriction--drug effects