Failure to complete apoptosis following neonatal hypoxia-ischemia manifests as "continuum" phenotype of cell death and occurs with multiple manifestations of mitochondrial dysfunction in rodent forebrain.
Northington, F J
Failure to complete apoptosis following neonatal hypoxia-ischemia manifests as "continuum" phenotype of cell death and occurs with multiple manifestations of mitochondrial dysfunction in rodent forebrain. [electronic resource] - Neuroscience Nov 2007 - 822-33 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural
0306-4522
10.1016/j.neuroscience.2007.06.060 doi
Analysis of Variance
Animals
Animals, Newborn
Apoptosis--physiology
Apoptotic Protease-Activating Factor 1--metabolism
Caspase 3--metabolism
Caspase 9--metabolism
Electron Transport Complex IV--metabolism
Gene Expression Regulation, Developmental--physiology
Hypoxia-Ischemia, Brain--pathology
Microscopy, Electron, Transmission--methods
Mitochondria--pathology
Neurons--diagnostic imaging
Prosencephalon--pathology
Rats
Time Factors
Ultrasonography
Failure to complete apoptosis following neonatal hypoxia-ischemia manifests as "continuum" phenotype of cell death and occurs with multiple manifestations of mitochondrial dysfunction in rodent forebrain. [electronic resource] - Neuroscience Nov 2007 - 822-33 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural
0306-4522
10.1016/j.neuroscience.2007.06.060 doi
Analysis of Variance
Animals
Animals, Newborn
Apoptosis--physiology
Apoptotic Protease-Activating Factor 1--metabolism
Caspase 3--metabolism
Caspase 9--metabolism
Electron Transport Complex IV--metabolism
Gene Expression Regulation, Developmental--physiology
Hypoxia-Ischemia, Brain--pathology
Microscopy, Electron, Transmission--methods
Mitochondria--pathology
Neurons--diagnostic imaging
Prosencephalon--pathology
Rats
Time Factors
Ultrasonography