Synergic CSE1L/CAS, TNFR-1, and p53 apoptotic pathways in combined interferon-gamma/adriamycin-induced apoptosis of Hep G2 hepatoma cells.
Jiang, M C
Synergic CSE1L/CAS, TNFR-1, and p53 apoptotic pathways in combined interferon-gamma/adriamycin-induced apoptosis of Hep G2 hepatoma cells. [electronic resource] - Journal of experimental & clinical cancer research : CR Mar 2007 - 91-9 p. digital
Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
0392-9078
Antineoplastic Agents--pharmacology
Apoptosis--drug effects
Carcinoma, Hepatocellular--metabolism
Cell Cycle--drug effects
Cell Line, Tumor
Cellular Apoptosis Susceptibility Protein--metabolism
Dose-Response Relationship, Drug
Doxorubicin--pharmacology
Drug Synergism
Etoposide--pharmacology
Humans
Interferon Regulatory Factor-1--genetics
Interferon-gamma--pharmacology
Liver Neoplasms--metabolism
Receptors, Tumor Necrosis Factor, Type I--metabolism
Signal Transduction--drug effects
Transfection
Tumor Suppressor Protein p53--metabolism
bcl-2-Associated X Protein--metabolism
bcl-Associated Death Protein--metabolism
Synergic CSE1L/CAS, TNFR-1, and p53 apoptotic pathways in combined interferon-gamma/adriamycin-induced apoptosis of Hep G2 hepatoma cells. [electronic resource] - Journal of experimental & clinical cancer research : CR Mar 2007 - 91-9 p. digital
Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
0392-9078
Antineoplastic Agents--pharmacology
Apoptosis--drug effects
Carcinoma, Hepatocellular--metabolism
Cell Cycle--drug effects
Cell Line, Tumor
Cellular Apoptosis Susceptibility Protein--metabolism
Dose-Response Relationship, Drug
Doxorubicin--pharmacology
Drug Synergism
Etoposide--pharmacology
Humans
Interferon Regulatory Factor-1--genetics
Interferon-gamma--pharmacology
Liver Neoplasms--metabolism
Receptors, Tumor Necrosis Factor, Type I--metabolism
Signal Transduction--drug effects
Transfection
Tumor Suppressor Protein p53--metabolism
bcl-2-Associated X Protein--metabolism
bcl-Associated Death Protein--metabolism