Evidence that beta-amyloid protein in Alzheimer's disease is not derived by normal processing.
Sisodia, S S
Evidence that beta-amyloid protein in Alzheimer's disease is not derived by normal processing. [electronic resource] - Science (New York, N.Y.) Apr 1990 - 492-5 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
0036-8075
10.1126/science.1691865 doi
Aged
Alzheimer Disease--metabolism
Amyloid--genetics
Amyloid beta-Peptides
Amyloid beta-Protein Precursor
Animals
Cell Membrane
Cells, Cultured
Cloning, Molecular
DNA, Recombinant
Glycosylation
Half-Life
Humans
Immunoblotting
Molecular Weight
Plasmids
Protein Precursors--genetics
Protein Processing, Post-Translational
Recombinant Fusion Proteins--metabolism
Substance P--genetics
Transfection
Evidence that beta-amyloid protein in Alzheimer's disease is not derived by normal processing. [electronic resource] - Science (New York, N.Y.) Apr 1990 - 492-5 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
0036-8075
10.1126/science.1691865 doi
Aged
Alzheimer Disease--metabolism
Amyloid--genetics
Amyloid beta-Peptides
Amyloid beta-Protein Precursor
Animals
Cell Membrane
Cells, Cultured
Cloning, Molecular
DNA, Recombinant
Glycosylation
Half-Life
Humans
Immunoblotting
Molecular Weight
Plasmids
Protein Precursors--genetics
Protein Processing, Post-Translational
Recombinant Fusion Proteins--metabolism
Substance P--genetics
Transfection