C-Raf antagonizes apoptosis induced by IFN-alpha in human lung cancer cells by phosphorylation and increase of the intracellular content of elongation factor 1A.

Lamberti, A

C-Raf antagonizes apoptosis induced by IFN-alpha in human lung cancer cells by phosphorylation and increase of the intracellular content of elongation factor 1A. [electronic resource] - Cell death and differentiation May 2007 - 952-62 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1350-9047

10.1038/sj.cdd.4402102 doi


Apoptosis--drug effects
Cell Line, Tumor
Humans
Immunoprecipitation
Interferon-alpha--pharmacology
Lung Neoplasms--metabolism
Oncogene Proteins--metabolism
Peptide Elongation Factor 1--metabolism
Phosphorylation--drug effects
Phosphoserine--metabolism
Phosphothreonine--metabolism
Proteasome Endopeptidase Complex--metabolism
Protein Binding--drug effects
Protein Processing, Post-Translational--drug effects
Protein Transport--drug effects
Proto-Oncogene Proteins c-raf--metabolism
RNA, Small Interfering--metabolism
Ubiquitin--metabolism